Enterococcus faecalis aggregation substance promotes opsonin-independent binding to human neutrophils via a complement receptor type 3-mediated mechanism

Natalie N. Vanek, Scott I. Simon, Karen Jacques-Palaz, M. Michele Mariscalco, Gary M. Dunny, Robert M. Rakita

Research output: Contribution to journalArticlepeer-review

63 Scopus citations

Fingerprint

Dive into the research topics of 'Enterococcus faecalis aggregation substance promotes opsonin-independent binding to human neutrophils via a complement receptor type 3-mediated mechanism'. Together they form a unique fingerprint.

Medicine & Life Sciences