Abstract
Three patients developed severe ovarian hyperstimulation syndrome (OHS) as a complication of ovarian hyperstimulation for in vitro fertilization. These patients presented with ovarian enlargement, vascular volume depletion, pleural effusions, and exudative ascites. A unique feature of the ascites in OHS was the markedly elevated renin concentration, the majority of which was prorenin. We speculate the renin-angiotensin system (RAS) may play a pathophysiologic role in the localized capillary leak that develops in OHS.
Original language | English (US) |
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Pages (from-to) | 427-429 |
Number of pages | 3 |
Journal | American Journal of Kidney Diseases |
Volume | 23 |
Issue number | 3 |
DOIs | |
State | Published - 1994 |
Keywords
- Ovarian hyperstimulation syndrome
- angiotensin
- ascites
- prorenin