Metabolic acidosis in restraint-associated cardiac arrest: A case series

John L. Hick, Stephen W. Smith, Michael T. Lynch

Research output: Contribution to journalArticlepeer-review

121 Scopus citations

Abstract

The mechanism of death in patients struggling against restraints remains a topic of debate. This article presents a series of five patients with restraint-associated cardiac arrest and profound metabolic acidosis. The lowest recorded pH was 6.25; this patient and three others died despite aggressive resuscitation. The survivor's pH was 6.46; this patient subsequently made a good recovery. Struggling against restraints may produce a lactic acidosis. Stimulant drugs such as cocaine may promote further metabolic acidosis and impair normal behavioral regulatory responses. Restrictive positioning of combative patients may impede appropriate respiratory compensation for this acidemia. Public safety personnel and emergency providers must be aware of the life threat to combative patients and be careful with restraint techniques. Further investigation of sedative agents and buffering therapy for this select patient group is suggested.

Original languageEnglish (US)
Pages (from-to)239-243
Number of pages5
JournalAcademic Emergency Medicine
Volume6
Issue number3
DOIs
StatePublished - Mar 1999

Keywords

  • Cardiac arrest
  • Heart arrest
  • Metabolic acidosis
  • Restraint - physical

Fingerprint Dive into the research topics of 'Metabolic acidosis in restraint-associated cardiac arrest: A case series'. Together they form a unique fingerprint.

Cite this